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Qing-Bai She

Formats
  1. Meeting Abstract (17)
  2. Journal Article (14)
Groups
  1. Department of Medicine (30)
  2. GSK Faculty (30)
  3. Solid Tumor Oncology Division (30)
  4. Sloan Kettering Institute Faculty (29)
  5. Breast Medicine Service (28)
  6. Molecular Pharmacology and Chemistry Program Faculty (28)

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People
  1. Neal Rosen (28)
  2. David Solit (20)
  3. Qing Ye (14)
  4. Kathryn O'reilly (5)
  5. Christine Pratilas (4)
  6. Jose Lobo (4)

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Years
  1. 2016 (1)
  2. 2012 (3)
  3. 2010 (6)
  4. 2009 (2)
  5. 2008 (4)
  6. 2007 (2)

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Publications
  1. Cancer Research (15)
  2. Cancer Cell (3)
  3. Clinical Cancer Research (3)
  4. BMC Cancer (1)
  5. Cancer Biology and Therapy (1)
  6. Cancer Discovery (1)

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Publishers
  1. American Association for Cancer Research (20)
  2. Cell Press (3)
  3. Nature Publishing Group (2)
  4. American Association for the Advancement of Science (1)
  5. Biomed Central Ltd (1)
  6. National Academy of Sciences (1)

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Results (31)

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  • Breast tumor cells with PI3K mutation or HER2 amplification are selectively dependent on Akt signaling
    Meeting Abstract

    She, Q. B.; Chandarlapaty, S.; Ye, Q.; Lobo, J.; Haskell, K.; Leander, K.; DeFeo-Jones, D.; Huber, H.; Rosen, N.
    Cancer Research (2009)

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  • Breast tumor cells with PI3K mutation or HER2 amplification are selectively addicted to Akt signaling
    Meeting Abstract

    She, Q. B.; Chandarlapaty, S.; Ye, Q.; Lobo, J.; Haskell, K. M.; Leander, K. R.; DeFeo-Jones, D.; Huber, H. E.; Rosen, N.
    Cancer Research (2009)

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  • Muc16 carboxyl portion expression alternates PI3K/Akt and EGFR/HER2/ERK signal pathways in human ovarian cells
    Meeting Abstract

    Ma, X.; Thapi, D.; Yan, X.; She, Q.; Rosales, N.; Spriggs, D. R.
    Cancer Research (2008)

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  • Coexistent PI3K mutation in human tumors is associated with decreased dependency on mutant KRAS and MEK/ERK signaling for transformation
    Meeting Abstract

    Halilovic, E.; She, Q.; Ye, Q.; Solit, D. B.; Rosen, N.
    Cancer Research (2008)

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  • Breast tumor cells with P13K mutation or HER2 amplification are selectively addicted to Akt signaling
    Journal Article

    She, Q. B.; Chandarlapaty, S.; Ye, Q.; Lobo, J.; Haskell, K. M.; Leander, K. R.; DeFeo-Jones, D.; Huber, H. E.; Rosen, N.
    PLoS ONE (2008)

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  • Models from experiments: Combinatorial drug perturbations of cancer cells
    Journal Article

    Nelander, S.; Wang, W.; Nilsson, B.; She, Q. B.; Pratilas, C.; Rosen, N.; Gennemark, P.; Sander, C.
    Molecular Systems Biology (2008)

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  • The PI3K/AKT and MAPK signaling cooperate to mediate translational initiation and cell survival in human tumors
    Meeting Abstract

    She, Q. B.; Ye, Q.; Zhen, W.; Solit, D.; Rosen, N.
    Cancer Research (2007)

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  • Poor prognosis in carcinoma is associated with a gene expression signature of aberrant PTEN tumor suppressor pathway activity
    Journal Article

    Saal, L. H.; Johansson, P.; Holm, K.; Gruvberger-Saal, S. K.; She, Q. B.; Maurer, M.; Koujak, S.; Ferrando, A. A.; MalmstroĢˆm, P.; Memeo, L.; more...
    Proceedings of the National Academy of Sciences of the United States of America (2007)

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  • Genetic and pharmacological inhibition of P13 kinase signaling in vivo sensitizes tumors to EGFR inhibitors and to taxol
    Meeting Abstract

    She, Q. B.; Ye, Q.; She, Y.; Solit, D. B.; Rosen, N.
    Cancer Research (2006)

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  • IGF-1 rescues rapamycin-induced cell growth inhibition
    Meeting Abstract

    O'reilly, K. E.; She, Q. B.; Solit, D. B.; Rosen, N.
    Cancer Research (2006)

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