Keywords: |
genetics; mouse; animal; metabolism; animals; mice; cell cycle; apoptosis; pathology; protein p53; physiology; cell transformation, neoplastic; gene expression regulation; drug antagonism; cell transformation; genomic instability; hematopoietic stem cells; fibroblast; fibroblasts; tumor suppressor protein p53; pluripotent stem cell; hematopoietic stem cell; cdkn2a protein, mouse; cyclin dependent kinase inhibitor 1a; cyclin dependent kinase inhibitor 2a; cyclin-dependent kinase inhibitor p16; cyclin-dependent kinase inhibitor p21; protein mdm2; proto-oncogene proteins c-mdm2; cdkn1a protein, human; mdm2 protein, human; nuclear reprogramming; induced pluripotent stem cells; mutagenesis, insertional
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