| Keywords: |
genetics; nonhuman; methodology; ovarian neoplasms; mouse; animal; metabolism; animals; mice; animal model; transgenic mouse; mice, transgenic; disease model; gene expression regulation; gene activation; genetic engineering; ovary tumor; tumor suppressor proteins; phosphatidylinositol 3,4,5 trisphosphate 3 phosphatase; pten phosphohydrolase; ovary carcinoma; short survey; ras protein; protein tyrosine phosphatase; ras proteins; pten protein, mouse; oncogene k ras; disease models, animal; tumor suppressor protein; cre recombinase; endometriosis; integrase; integrases; protein-tyrosine-phosphatase; gynecologic disease
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