Keywords: |
cancer survival; gene mutation; cisplatin; doxorubicin; fluorouracil; cancer growth; pathophysiology; antineoplastic agent; letter; proteins; cell survival; melanoma; reverse transcription polymerase chain reaction; apoptosis; gene expression; etoposide; down-regulation; cancer cell culture; in vitro study; cell line, tumor; vinblastine; time factors; in situ hybridization; gene expression regulation, neoplastic; intracellular signaling peptides and proteins; tumor suppressor proteins; drug response; sample size; northern blotting; apoptotic protease activating factor 1; apoptotic protease-activating factor 1
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