Keywords: |
signal transduction; unclassified drug; gene mutation; mutation; janus kinase 2; cancer growth; protein function; neoplasms; cell death; stat3 protein; signaling; phosphatase; protein protein interaction; epidermal growth factor receptor; gene locus; editorial; receptor, epidermal growth factor; phosphorylation; protein tyrosine kinase inhibitor; gene number; oncogene; tumor suppressor gene; regulatory mechanism; colon cancer; stat3 transcription factor; receptor; tumor suppressor; bcr abl protein; protein tyrosine phosphatase; protein tyrosine phosphatase receptor d; protein tyrosine phosphatase receptor t; gene silencing; heterozygosity loss; protein dephosphorylation; receptor-like protein tyrosine phosphatases, class 2
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