Keywords: |
signal transduction; protein expression; oncoprotein; genetics; proto-oncogene proteins; pathogenesis; cancer growth; nonhuman; mouse; dna damage; cell survival; cell cycle; dna repair; proto oncogene; biology; editorial; transgenic mouse; polo like kinase 1; genomic instability; carcinogenicity; cellular distribution; clinical pathway; cell cycle checkpoint; dna damage response; chronic lymphatic leukemia; tumor promoter; leukemia, lymphocytic, chronic, b-cell; murine model; pleiotropy; chromosome segregation; multiprotein complex; proto-oncogenes; cell cycle protein 20; protein mad2; tcl1a protein, human; m phase cell cycle checkpoint; humans; human; wild type mouse; proto oncogene protein
|