Keywords: |
epidermal growth factor; human cell; overall survival; exon; genetics; mutation; erlotinib; monotherapy; note; progression free survival; apoptosis; pharmacodynamics; carcinoma, non-small-cell lung; lung neoplasms; epidermal growth factor receptor; cetuximab; protein tyrosine kinase inhibitor; lung tumor; xenograft; gefitinib; tumor cell; binding site; dimerization; therapy resistance; mammalian target of rapamycin inhibitor; ligand binding; egfr protein, human; pharmacology; epidermal growth factor receptor kinase inhibitor; scatter factor receptor; non small cell lung cancer; neu differentiation factor; clinical outcome; egfr signaling; erbb receptors; afatinib; dacomitinib; humans; human; osimertinib
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