Keywords: |
gene mutation; genetics; erlotinib; note; metabolism; proto oncogene; enzyme inhibition; ovary cancer; breast cancer; gene amplification; epidermal growth factor receptor; epidermal growth factor receptor 2; lung cancer; editorial; receptor, epidermal growth factor; cetuximab; monoclonal antibody; panitumumab; prostate cancer; prostatic neoplasms; antibodies, monoclonal; colon cancer; prostate tumor; phosphatidylinositol 3,4,5 trisphosphate 3 phosphatase; gefitinib; ras protein; receptor, erbb-2; conformational transition; trastuzumab; genes, erbb-2; b raf kinase; lapatinib; pertuzumab
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