Risk of disease progression in low-risk MDS is linked to distinct epigenetic subtypes - To the editor Journal Article


Authors: Qin, T.; Sotzen, J.; Rampal, R. K.; Rapaport, F. T.; Levine, R. L.; Klimek, V.; Nimer, S. D.; Figueroa, M. E.
Article Title: Risk of disease progression in low-risk MDS is linked to distinct epigenetic subtypes - To the editor
Keywords: signal transduction; mitogen activated protein kinase; clinical article; controlled study; gene mutation; gene sequence; promoter region; mutation; dna replication; letter; gene; cell cycle; protein protein interaction; cohort analysis; intron; gene frequency; hemoglobin; hemoglobin blood level; chronic myeloid leukemia; protein p53; dna methylation; risk; cytokine; myelodysplastic syndrome; epigenetics; cpg island; mammalian target of rapamycin; excision repair; receptor; wnt protein; mtor signaling; cytopenia; disease exacerbation; oxidative phosphorylation; tet2 gene; enhancer region; transcription factor runx1; wnt signaling; transfusion; mapk signaling; human; priority journal; runx1 gene; mutation burden; bisulfite sequencing; enhanced reduced representation bisulfite sequencing; p53 signaling
Journal Title: Leukemia
Volume: 33
Issue: 11
ISSN: 0887-6924
Publisher: Nature Publishing Group  
Date Published: 2019-11-01
Start Page: 2753
End Page: 2757
Language: English
DOI: 10.1038/s41375-019-0518-5
PUBMED: 31332268
PROVIDER: scopus
PMCID: PMC6842085
DOI/URL:
Notes: Source: Scopus
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MSK Authors
  1. Virginia Klimek
    122 Klimek
  2. Raajit Kumar Rampal
    158 Rampal
  3. Ross Levine
    540 Levine
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