Keywords: |
aged; unclassified drug; gene mutation; genetics; mutation; cancer risk; note; metabolism; dna repair; genetic predisposition to disease; breast cancer; ubiquitin protein ligase; morbidity; tumor markers, biological; genetic association; editorial; enzymology; protein serine threonine kinase; breast neoplasms; tumor marker; population research; protein-serine-threonine kinases; family history; breast tumor; checkpoint kinase 2; genetic predisposition; ubiquitin-protein ligases; germ-line mutation; brap protein, human
|