| Keywords: |
controlled study; gene mutation; gene deletion; mutation; proto-oncogene proteins; nonhuman; mouse; animal; mice; animal tissue; cells, cultured; proto oncogene; bone marrow; melanocyte; melanocytes; embryo; mice, mutant strains; embryo development; protein tyrosine kinase; mice, inbred c57bl; gene expression regulation; dna; gene rearrangement; messenger rna; protein synthesis; gestational age; ligand; lung; pigmentation disorders; receptor protein-tyrosine kinases; mesoderm; mesenchyme; neural tube; melanogenesis; gene expression regulation, enzymologic; mice, inbred c3h; mast cell; mast cells; receptor protein; melanins; digestive system; somite; priority journal; article; support, u.s. gov't, p.h.s.; dermatome; c-kit receptor; proto-oncogene protein c-kit; receptors, colony-stimulating factor; expression mutation; negative and positive control elements; w-sash
|
