Modeling SF3B1 mutations in cancer: Advances, challenges, and opportunities Journal Article


Authors: Inoue, D.; Abdel-Wahab, O.
Article Title: Modeling SF3B1 mutations in cancer: Advances, challenges, and opportunities
Abstract: In this issue of Cancer Cell, Obeng et al. identify the consequences of expressing the most common mutation in the spliceosomal gene SF3B1 on hematopoiesis. The knockin mouse model described represents a valuable tool to dissect the effects of SF3B1 mutations on transformation, splicing, and less well-characterized functions of SF3B1. © 2016 Elsevier Inc.
Journal Title: Cancer Cell
Volume: 30
Issue: 3
ISSN: 1535-6108
Publisher: Cell Press  
Date Published: 2016-09-12
Start Page: 371
End Page: 373
Language: English
DOI: 10.1016/j.ccell.2016.08.013
PROVIDER: scopus
PUBMED: 27622329
PMCID: PMC5569387
DOI/URL:
Notes: Short Survey -- Export Date: 6 December 2016 -- Source: Scopus
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MSK Authors
  1. Daichi   Inoue
    16 Inoue