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David Solit

Formats
  1. Meeting Abstract (391)
  2. Journal Article (366)
  3. Editorial (9)
  4. Book Section (4)
  5. Research Letter (4)
  6. Review (4)
  7. Correspondence (2)
  8. Guidelines (2)
  9. Conference Paper (1)
Groups
  1. Department of Medicine (783)
  2. GSK Faculty (783)
  3. Genitourinary Oncology Service (783)
  4. Human Oncology and Pathogenesis Program (783)
  5. Solid Tumor Oncology Division (783)
  6. Department of Pathology and Laboratory Medicine (510)

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People
  1. Michael Berger (317)
  2. Hikmat Al-Ahmadie (186)
  3. Gopakumar Iyer (184)
  4. Neal Rosen (155)
  5. Nikolaus Schultz (150)
  6. Dean Bajorin (130)

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Years
  1. 2025 (17)
  2. 2024 (38)
  3. 2023 (39)
  4. 2022 (44)
  5. 2021 (47)
  6. 2020 (59)

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Publications
  1. Journal of Clinical Oncology (163)
  2. Cancer Research (99)
  3. Clinical Cancer Research (77)
  4. Laboratory Investigation (42)
  5. Modern Pathology (37)
  6. Cancer Discovery (24)

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Publishers
  1. American Association for Cancer Research (219)
  2. American Society of Clinical Oncology (181)
  3. Nature Publishing Group (97)
  4. Nature Research (39)
  5. Elsevier Inc. (31)
  6. Oxford University Press (29)

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Results (783)


Citation style sort is year ascending, and then by author or title. Author ascending is default.
  • The Akt protein kinase is a direct target of ansamycin antibiotics
    Meeting Abstract

    Basso, A. D.; Solit, D.; Rosen, N.
    Proceedings of the American Association for Cancer Research Annual Meeting (2002)

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  • Ansamycins inactivate AKT and enhances the anti-tumor effects of paclitaxel
    Meeting Abstract

    Solit, D.; Basso, A.; Munster, P.; Scher, H.; Rosen, N.
    European Journal of Cancer (2002)

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  • 17-Allylamino-17-demethoxygeldanamycin induces the degradation of androgen receptor and HER-2/neu and inhibits the growth of prostate cancer xenografts
    Journal Article

    Solit, D. B.; Zheng, F. F.; Drobnjak, M.; Mùˆnster, P. N.; Higgins, B.; Verbel, D.; Heller, G.; Tong, W.; Cordon-Cardo, C.; Agus, D. B.; more...
    Clinical Cancer Research (2002)

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  • Ansamycin antibiotics inhibit Akt activation and cyclin D expression in breast cancer cells that overexpress HER2
    Journal Article

    Basso, A. D.; Solit, D. B.; Munster, P. N.; Rosen, N.
    Oncogene (2002)

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  • Akt forms an intracellular complex with heat shock protein 90 (Hsp90) and Cdc37 and is destabilized by inhibitors of Hsp90 function
    Journal Article

    Basso, A. D.; Solit, D. B.; Chiosis, G.; Giri, B.; Tsichlis, P.; Rosen, N.
    Journal of Biological Chemistry (2002)

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  • Modulation of Hsp90 function by ansamycins enhances chemotherapy induced apoptosis in breast cancer cell lines in an RB and schedule dependent manner
    Meeting Abstract

    Munster, P. N.; Basso, A. D.; Solit, D.; Norton, L.; Scher, H.; Rosen, N.
    Proceedings of the American Association for Cancer Research Annual Meeting (2001)

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  • Ansamycins inhibit Akt activation and induce the 20S proteasomal degradation of Akt protein
    Meeting Abstract

    Basso, A. D.; Solit, D. B.; Rosen, N.
    Clinical Cancer Research (2001)

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  • 17-Allylamino-geldanamycin causes AKT inactivation and enhances the anti-tumor effects of paclitaxel
    Meeting Abstract

    Solit, D. B.; Basso, A. D.; Munster, P. N.; Norton, L.; Scher, H. I.; Rosen, N.
    Clinical Cancer Research (2001)

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  • Phase I trial of 17AAG (17-allylamino) 17-demethoxygeldanamycin (17AAG) in patients (Pts) with advanced cancer
    Meeting Abstract

    Scher, H. I.; Solit, D. B.; Munster, P. N.; Tong, W. P.; Lawrence, S.; Kenneson, K.; Larson, S.; De La Cruz, A.; Reuter, V.; Rosen, N.
    Clinical Cancer Research (2001)

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  • HER-kinase-directed therapy of prostate cancer
    Journal Article

    Solit, D. B.; Agus, D. B.
    Prostate Journal (2001)

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