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Mark M Moasser

  1. "Genitourinary Oncology Service"
Formats
  1. Meeting Abstract (7)
  2. Journal Article (3)
Groups
  1. Department of Medicine (10)
  2. Genitourinary Oncology Service (10)
  3. Solid Tumor Oncology Division (10)
  4. GSK Faculty (9)
  5. Breast Medicine Service (8)
  6. Molecular Pharmacology and Chemistry Program Faculty (8)

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People
  1. Neal Rosen (8)
  2. David Solit (7)
  3. Howard Scher (4)
  4. Qing-Bai She (4)
  5. Andrea Basso (3)
  6. Clifford Hudis (2)

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Years
  1. 2004 (1)
  2. 2003 (5)
  3. 2002 (1)
  4. 2001 (1)
  5. 1998 (1)
  6. 1995 (1)
Publications
  1. Proceedings of the American Association for Cancer Research Annual Meeting (4)
  2. Clinical Cancer Research (3)
  3. Cancer (1)
  4. Cancer Research (1)
  5. Prostate Cancer and Prostatic Diseases (1)
Publishers
  1. American Association for Cancer Research (8)
  2. Nature Publishing Group (1)
  3. Wiley Blackwell (1)

Results (10)

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  • Restoration of PTEN-expression in tumor cells causes them to depend on EGFR for suppression
    Meeting Abstract

    She, Q.; Moasser, M. M.; Ye, Q.; O'reilly, K.; Solit, D.; Rosen, N.
    Cancer Research (2004)

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  • Pulsatile administration of the EGF receptor inhibitor Iressa (ZD1839) is significantly more effective than continuous dosing for sensitizing tumors to taxol
    Meeting Abstract

    Solit, D. B.; She, Y.; Moasser, M. M.; Hudis, C. A.; Kris, M. G.; Scher, H. I.; Rosen, N.; Sirotnak, F. M.
    Proceedings of the American Association for Cancer Research Annual Meeting (2003)

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  • Response and resistance to gefitinib (ZD1839, Iressa) in HER overexpressing tumor cells is mediated through the PI3K/Akt pathway
    Meeting Abstract

    She, Q. B.; Solit, D.; Basso, A.; Rosen, N.; Moasser, M. M.
    Proceedings of the American Association for Cancer Research Annual Meeting (2003)

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  • Pulsatile administration of the EGF receptor inhibitor gefitinib (Iressa, ZD 1839) is significantly more effective than continuous dosing for sensitizing tumors to Taxol
    Meeting Abstract

    Solit, D. B.; She, Y.; Moasser, M.; Hudis, C.; Kris, M.; Scher, H.; Rosen, N.; Sirotnak, F. M.
    Clinical Cancer Research (2003)

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  • Cyclin D expression and G1 progression are dependent on MEK-MAP kinase activity in tumor cells with B-Raf mutation but not in breast cancer cells with wild type Ras
    Meeting Abstract

    Sawai, A.; Basso, A. D.; Solit, D. B.; She, Q. B.; Grbovic, O.; Pratilas, C.; Chapman, P.; Moasser, M. M.; Rosen, N.
    Clinical Cancer Research (2003)

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  • Resistance to gefitinib in PTEN-Null HER-overexpressing tumor cells can be overcome through restoration of PTEN function or pharmacologic modulation of constitutive phosphatidylinositol 3′-kinase/Akt pathway signaling
    Journal Article

    She, Q. B.; Solit, D.; Basso, A.; Moasser, M. M.
    Clinical Cancer Research (2003)

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  • Ras signaling is not required for transformed growth but does mediate the angiogenic properties of MCF-7 cells
    Meeting Abstract

    Moasser, M. M.; Solit, D.; Costa, C.; Rafii, S.; Rosen, N.
    Proceedings of the American Association for Cancer Research Annual Meeting (2002)

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  • Farnesyl:protein transferase inhibitors as potential agents for the management of human prostate cancer
    Journal Article

    Sepp-Lorenzino, L.; Tjaden, G.; Moasser, M. M.; Timaul, N.; Ma, Z.; Kohl, N. E.; Gibbs, J. B.; Oliff, A.; Rosen, N.; Scher, H. I.
    Prostate Cancer and Prostatic Diseases (2001)

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  • Farnesyl: Protein transferase inhibitors (FTIs): Potential chemotypes for the management of human prostate cancer
    Meeting Abstract

    Sepp-Lorenzino, L.; Tjaden, G.; Timaul-Holmes, M.; Moasser, M.; Chou, J. L.; Balog, A.; Su, D. S.; Danishefsky, S. J.; Kohl, N.; Oliff, A.; more...
    Proceedings of the American Association for Cancer Research Annual Meeting (1998)

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  • all‐trans retinoic acid for treating germ cell tumors: In vitro activity and results of a phase II trial
    Journal Article

    Moasser, M. M.; Motzer, R. J.; Khoo, K. S.; Lyn, P.; Murphy, B. A.; Bosl, G. J.; Dmitrovsky, E.
    Cancer (1995)

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